Postherpetic trigeminal neuralgia
ICD-10 B02.22 is a billable code used to indicate a diagnosis of postherpetic trigeminal neuralgia.
Postherpetic trigeminal neuralgia (PHTN) is a chronic pain condition that occurs following an outbreak of herpes zoster (shingles), which is caused by the reactivation of the varicella-zoster virus (VZV). This condition is characterized by severe, persistent facial pain in the distribution of the trigeminal nerve, often described as burning, stabbing, or electric shock-like sensations. The pain can be triggered by light touch or temperature changes, significantly impacting the patient's quality of life. PHTN typically develops in older adults, particularly those with a history of shingles, and is more common in individuals with weakened immune systems. The pain may persist for months or even years after the shingles rash has healed, making early diagnosis and management crucial. Treatment options include medications such as anticonvulsants, antidepressants, and topical agents, as well as interventional procedures in refractory cases. Understanding the pathophysiology of PHTN, including the role of nerve damage and inflammation, is essential for effective management and coding.
Detailed history of pain, including onset, duration, and triggers; neurological examination findings.
Patients presenting with facial pain post-herpes zoster; evaluation of pain management strategies.
Ensure documentation reflects the chronic nature of the pain and any treatments attempted.
Comprehensive pain assessments, treatment plans, and response to therapies.
Management of refractory pain in patients with PHTN; interventional procedures for pain relief.
Document all modalities used for pain management to support coding and billing.
Used for patients with refractory PHTN pain.
Document the indication for the procedure and the patient's pain history.
Pain management specialists should ensure comprehensive pain assessments are included.
Postherpetic trigeminal neuralgia is primarily caused by the reactivation of the varicella-zoster virus, which leads to inflammation and damage to the trigeminal nerve following a shingles outbreak.
