Drug or chemical induced diabetes mellitus with mild nonproliferative diabetic retinopathy without macular edema (bilateral)
ICD-10 E09.3293 is a billable code used to indicate a diagnosis of drug or chemical induced diabetes mellitus with mild nonproliferative diabetic retinopathy without macular edema (bilateral).
E09.3293 refers to diabetes mellitus that has been induced by drugs or chemicals, characterized by the presence of mild nonproliferative diabetic retinopathy without macular edema in both eyes. This condition arises when certain medications, such as corticosteroids or antipsychotics, lead to glucose intolerance or insulin resistance, resulting in elevated blood glucose levels. The mild nonproliferative diabetic retinopathy indicates early changes in the retina due to diabetes, including microaneurysms and retinal hemorrhages, but without the presence of macular edema, which is a more severe complication. Management of this condition typically involves monitoring blood glucose levels, adjusting medications, and possibly using insulin therapy to maintain glycemic control. Regular eye examinations are crucial to monitor retinal changes and prevent progression to more severe diabetic retinopathy.
Detailed medication history, HbA1c levels, and management plans.
Patients presenting with elevated blood glucose levels after starting new medications.
Ensure clear documentation of the relationship between drug use and diabetes onset.
Retinal examination findings, including details on the severity of retinopathy.
Routine eye exams for diabetic patients to monitor for retinopathy.
Document the absence of macular edema clearly to support coding.
Used for follow-up visits for diabetes management.
Document patient history, examination findings, and management plan.
Endocrinologists should ensure comprehensive documentation of diabetes management.
Common medications that can induce diabetes include corticosteroids, thiazide diuretics, and certain antipsychotics. It is important to document any medication changes that may have contributed to the onset of diabetes.
