Acquired keratosis [keratoderma] palmaris et plantaris
ICD-10 L85.1 is a billable code used to indicate a diagnosis of acquired keratosis [keratoderma] palmaris et plantaris.
Acquired keratosis palmaris et plantaris, classified under ICD-10 code L85.1, is a dermatological condition characterized by the thickening of the skin on the palms of the hands and the soles of the feet. This condition often presents as rough, scaly patches that can vary in color from yellowish to brown. The thickening occurs due to an increase in keratin, a protein that protects the skin, often as a response to friction, pressure, or irritation. The condition can be associated with various factors, including occupational exposure, genetic predisposition, or underlying systemic diseases. Disease progression may lead to discomfort, pain, or secondary infections if the skin becomes fissured. Diagnosis typically involves a clinical examination, and in some cases, a biopsy may be performed to rule out other skin disorders. It is essential for healthcare providers to differentiate acquired keratosis from other keratodermas, such as palmoplantar keratoderma, which may have a hereditary component. Proper identification and management are crucial to prevent complications and improve patient quality of life.
Standard ICD-10-CM documentation requirements apply
Various clinical presentations within this specialty area
Follow specialty-specific billing guidelines
Standard ICD-10-CM documentation requirements apply
Various clinical presentations within this specialty area
Follow specialty-specific billing guidelines
L85.1 specifically covers acquired keratosis palmaris et plantaris, which includes conditions resulting in thickened skin on the palms and soles due to acquired factors such as friction or pressure. It does not cover hereditary keratodermas or other forms of keratosis.
L85.1 should be used when the keratosis is acquired and localized to the palms and soles. It is important to use this code when the condition is clearly linked to external factors rather than genetic predisposition.
Documentation should include a thorough clinical examination, patient history detailing exposure to irritants or friction, and any treatments attempted. Photographic evidence may also be beneficial in supporting the diagnosis.
