Osteonecrosis due to drugs of right carpus
ICD-10 M87.137 is a billable code used to indicate a diagnosis of osteonecrosis due to drugs of right carpus.
Osteonecrosis, also known as avascular necrosis, is a condition characterized by the death of bone tissue due to a lack of blood supply. In the case of M87.137, the osteonecrosis is specifically attributed to the use of certain drugs, which can compromise blood flow to the bones. The right carpus, consisting of eight small bones in the wrist, is particularly susceptible to this condition due to its anatomical structure and blood supply. Commonly implicated drugs include corticosteroids and bisphosphonates, which can lead to increased intraosseous pressure and subsequent ischemia. Patients may present with pain, swelling, and limited range of motion in the wrist, and if left untreated, the condition can progress to joint collapse and severe disability. Diagnosis typically involves imaging studies such as MRI or X-rays, which can reveal characteristic changes in bone density and structure. Treatment options may include medication adjustments, physical therapy, or surgical interventions such as core decompression or joint replacement, depending on the severity of the condition.
Detailed history of drug use, imaging results, and treatment plans.
Patients presenting with wrist pain after corticosteroid therapy.
Ensure clear documentation of the relationship between drug use and osteonecrosis.
Comprehensive medication history and assessment of joint function.
Patients on long-term corticosteroids for autoimmune conditions developing wrist pain.
Document underlying conditions that may contribute to osteonecrosis.
Used for pain management in patients with osteonecrosis.
Document the joint involved and the reason for the procedure.
Orthopedic specialists should ensure accurate documentation of the joint and condition.
Common drugs include corticosteroids, bisphosphonates, and certain chemotherapy agents. It's important to document any relevant medication history when coding.