Other transitory neonatal disorders of thyroid function, not elsewhere classified
ICD-10 P72.2 is a billable code used to indicate a diagnosis of other transitory neonatal disorders of thyroid function, not elsewhere classified.
P72.2 refers to a category of transient neonatal thyroid dysfunctions that are not classified under other specific codes. These disorders can manifest as either hyperthyroidism or hypothyroidism in newborns, often resulting from maternal factors such as iodine deficiency, autoimmune disorders, or exposure to antithyroid medications during pregnancy. The conditions are typically self-limiting, resolving within weeks to months as the newborn's endocrine system matures. Clinical manifestations may include lethargy, poor feeding, hypotonia, or, conversely, irritability and increased activity levels. Diagnosis is primarily based on serum thyroid hormone levels, including TSH and free T4, and may require follow-up testing to ensure resolution. Understanding these conditions is crucial for timely intervention and management to prevent potential long-term neurodevelopmental impacts.
Detailed records of thyroid function tests, clinical observations, and follow-up assessments are essential.
Neonates presenting with lethargy or irritability, requiring thyroid function evaluation in the NICU.
Ensure clarity in documentation regarding the transient nature of the disorder and resolution timelines.
Pediatric follow-up notes should include growth parameters and developmental assessments.
Pediatric visits for monitoring thyroid function in infants previously diagnosed with transient disorders.
Consider the long-term developmental impact and ensure appropriate referrals if needed.
Used to evaluate thyroid function in newborns suspected of having thyroid dysfunction.
Document the indication for testing and results.
Neonatologists should ensure that testing is timely and results are communicated effectively.
Common causes include maternal autoimmune thyroid disease, exposure to antithyroid medications during pregnancy, and iodine deficiency. These factors can lead to temporary alterations in the newborn's thyroid function, which typically resolves as the infant matures.