Von Willebrand disease, type 2, unspecified
ICD-10 D68.029 is a billable code used to indicate a diagnosis of von willebrand disease, type 2, unspecified.
Von Willebrand disease (VWD) is a genetic bleeding disorder caused by a deficiency or dysfunction of von Willebrand factor (VWF), a protein crucial for platelet adhesion and aggregation. Type 2 VWD is characterized by a qualitative defect in VWF, leading to varying degrees of bleeding symptoms. Patients may experience easy bruising, prolonged bleeding from cuts, heavy menstrual periods, and spontaneous bleeding episodes. The severity of symptoms can vary widely among individuals, and the diagnosis is often confirmed through laboratory tests that assess VWF levels and function. Type 2 VWD is further classified into subtypes (2A, 2B, 2M, and 2N), but when unspecified, it indicates a general diagnosis without further classification. Management typically involves desmopressin administration, which stimulates the release of VWF, and in some cases, factor replacement therapy may be necessary. Understanding the nuances of this condition is essential for accurate coding and appropriate patient management.
Comprehensive documentation of bleeding history, laboratory test results, and treatment plans.
Patients presenting with unexplained bleeding, easy bruising, or a family history of bleeding disorders.
Ensure accurate classification of VWD type and thorough documentation of any genetic testing performed.
Detailed family history and genetic testing results to confirm diagnosis.
Patients with a family history of bleeding disorders seeking genetic counseling.
Document the implications of genetic findings on treatment and family planning.
Used for laboratory tests to confirm VWD diagnosis.
Document the reason for blood collection and any relevant clinical history.
Ensure that the specialty performing the test is noted in the documentation.
Von Willebrand disease type 2 is a bleeding disorder caused by a qualitative defect in von Willebrand factor, leading to impaired platelet function and increased bleeding risk.
