(Idiopathic) normal pressure hydrocephalus
ICD-10 G91.2 is a billable code used to indicate a diagnosis of (idiopathic) normal pressure hydrocephalus.
Idiopathic normal pressure hydrocephalus (NPH) is a neurological condition characterized by an accumulation of cerebrospinal fluid (CSF) in the brain's ventricles, leading to ventricular enlargement without an increase in intracranial pressure. This condition typically presents in older adults and is often associated with a classic triad of symptoms: gait disturbance, cognitive dysfunction, and urinary incontinence. The exact etiology of idiopathic NPH remains unclear, but it is thought to involve a combination of factors including age-related changes in CSF dynamics and potential neurodegenerative processes. Diagnosis is primarily clinical, supported by neuroimaging findings that reveal ventricular enlargement. Treatment often involves the surgical placement of a ventriculoperitoneal shunt to drain excess CSF, which can lead to significant improvement in symptoms if performed early. However, the response to treatment can vary, and not all patients experience the same degree of benefit.
Detailed neurological examination findings, neuroimaging reports, and symptom progression.
Patients presenting with gait disturbances, cognitive decline, and urinary incontinence.
Ensure that all symptoms are documented clearly and that neuroimaging supports the diagnosis.
Surgical notes, pre-operative assessments, and post-operative follow-up documentation.
Patients undergoing ventriculoperitoneal shunt placement for NPH.
Document the rationale for surgical intervention and any complications or outcomes.
Used for patients diagnosed with idiopathic NPH requiring surgical intervention.
Surgical consent, pre-operative assessments, and post-operative follow-up notes.
Neurosurgeons must document the rationale for surgery and any complications.
The key symptoms include gait disturbance, cognitive decline, and urinary incontinence, often referred to as the classic triad.
Diagnosis is based on clinical evaluation, the presence of the classic triad of symptoms, and neuroimaging that shows ventricular enlargement without increased intracranial pressure.
