Idiopathic aseptic necrosis of unspecified tibia
ICD-10 M87.063 is a billable code used to indicate a diagnosis of idiopathic aseptic necrosis of unspecified tibia.
Idiopathic aseptic necrosis of the tibia is a condition characterized by the death of bone tissue due to a lack of blood supply, without an identifiable cause. This condition can lead to significant pain, swelling, and functional impairment of the affected limb. The tibia, being a weight-bearing bone, is particularly susceptible to the effects of osteonecrosis, which can result in joint dysfunction and long-term disability if not addressed promptly. Patients may present with symptoms such as localized pain that worsens with activity, tenderness on palpation, and possible swelling. Diagnosis typically involves imaging studies, including X-rays and MRI, which can reveal changes in bone density and structure indicative of necrosis. Treatment options may vary from conservative management, such as physical therapy and pain relief, to surgical interventions like core decompression or osteotomy, depending on the severity of the condition and the patient's overall health. Understanding the idiopathic nature of this condition is crucial, as it often requires a multidisciplinary approach for effective management.
Detailed clinical notes, imaging results, and treatment plans.
Patients presenting with knee or ankle pain, history of trauma, or chronic pain without clear etiology.
Ensure clear documentation of the idiopathic nature and any differential diagnoses considered.
Functional assessments, treatment goals, and progress notes.
Rehabilitation following surgical intervention for necrosis or conservative management.
Document functional limitations and response to therapy to support ongoing treatment.
Used for pain management in patients with necrosis-related joint effusion.
Document the indication for the procedure and any imaging findings.
Orthopedic specialists should ensure that the procedure is justified based on clinical findings.
The primary cause is unknown, but it is believed to involve a disruption of blood supply to the bone, leading to necrosis without any identifiable underlying condition.
