Chronic nephritic syndrome with dense deposit disease
ICD-10 N03.6 is a billable code used to indicate a diagnosis of chronic nephritic syndrome with dense deposit disease.
Chronic nephritic syndrome with dense deposit disease is a specific form of glomerulonephritis characterized by the presence of dense deposits in the glomerular basement membrane. This condition is often associated with significant proteinuria, hematuria, and hypertension. Patients may present with symptoms of nephrotic syndrome, including edema and foamy urine due to high levels of protein loss. The pathophysiology involves the deposition of complement component C3 and immunoglobulin in the kidney, leading to inflammation and damage to the glomeruli. Renal biopsy findings typically reveal a characteristic pattern of dense deposits and may show a membranoproliferative glomerulonephritis pattern. Management of this condition often requires a multidisciplinary approach, including nephrology for monitoring renal function, managing hypertension, and addressing proteinuria, which may involve the use of angiotensin-converting enzyme (ACE) inhibitors or corticosteroids. Regular follow-up is essential to monitor for progression to end-stage renal disease.
Detailed clinical notes on renal function, laboratory results, and treatment plans.
Management of chronic kidney disease, monitoring of proteinuria, and treatment of hypertension.
Ensure accurate coding of all associated conditions and regular updates on patient status.
Comprehensive reports on renal biopsy findings, including histological analysis.
Evaluation of renal biopsies for glomerular diseases.
Pathology reports must clearly indicate the presence of dense deposits and any other relevant findings.
Used when a renal biopsy is performed to confirm diagnosis.
Pathology report detailing findings and indication for biopsy.
Nephrologists must ensure that the biopsy is justified based on clinical presentation.
Dense deposit disease is a specific type of chronic nephritic syndrome that indicates a unique pathophysiological process involving complement dysregulation, leading to distinct clinical and histological features.